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Mutualism with our intestinal microbiota is a prerequisite for healthy existence. This requires physical separation of the majority of the microbiota from the host (by secreted antimicrobials, mucus, and the intestinal epithelium) and active immune control of the low numbers of microbes that overcome these physical and chemical barriers, even in healthy individuals. In this review, we address how B-cell responses to members of the intestinal microbiota form a robust network with mucus, epithelial integrity, follicular helper T cells, innate immunity, and gut-associated lymphoid tissues to maintain host-microbiota mutualism.

Original publication

DOI

10.1111/imr.12179

Type

Journal article

Journal

Immunol Rev

Publication Date

07/2014

Volume

260

Pages

50 - 66

Keywords

B lymphocytes, IgA, gut-associated lymphoid tissue, innate immunity, intestinal barrier, microbiota, Animals, Autophagy, B-Lymphocyte Subsets, Cellular Microenvironment, Endoplasmic Reticulum Stress, Fatty Acids, Histone Deacetylases, Host-Pathogen Interactions, Humans, IgA Deficiency, Immune System, Immunity, Innate, Immunity, Mucosal, Immunoglobulin A, Immunologic Deficiency Syndromes, Intestinal Mucosa, Intestines, Microbiota, Mucus, Peyer's Patches, Symbiosis, T-Lymphocyte Subsets