Cookies on this website

We use cookies to ensure that we give you the best experience on our website. If you click 'Accept all cookies' we'll assume that you are happy to receive all cookies and you won't see this message again. If you click 'Reject all non-essential cookies' only necessary cookies providing core functionality such as security, network management, and accessibility will be enabled. Click 'Find out more' for information on how to change your cookie settings.

Kate Attfield

Collaborators

Kate Attfield

Principal Investigator

Physical and mental disability caused by neuroinflammation is a common clinical occurrence in many diseases, including multiple sclerosis (MS), Alzheimer’s disease and schizophrenia. However, the cause(s) of this inflammation and the devastating consequences that ensue are poorly defined, as reflected by a lack of effective treatments.

As demonstrated by our previous work, with a particular focus on multiple sclerosis, we have shown how studying individual cells directly from patient blood or mapping biological events across the diseased brain in post-mortem tissue, can expose an intricate network of neuroinflammatory processes, which change through time and space. With evolving technologies, our research team seeks to understand how the immune system and resident cells of the central nervous system orchestrate these disease processes.