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Publications

Non-Surgical Periodontal Therapy Plus Short-Term Antibiotic Treatment May Improve Clinical Disease Activity: A Pilot Study in Difficult to Treat Rheumatoid Arthritis

(2019), Arthritis & Rheumatology

Alternative splicing of tenascin-C modulates cell-matrix interactions during inflammation and disease

(2019), MOLECULAR BIOLOGY OF THE CELL

Treatment of severe periodontitis may improve clinical disease activity in otherwise treatment-refractory rheumatoid arthritis

MIDWOOD KIM., (2019), Rheumatology

Citrullination of fibronectin alters integrin clustering and focal adhesion stability promoting stromal cell invasion.

Stefanelli VL. et al, (2019), Matrix Biol

Response to: 'Potential roles for tenascin in (very) early diagnosis and treatment of rheumatoid arthritis' by Cutolo et al.

Aungier S. et al, (2019), Ann Rheum Dis

Human rhinovirus infection of airway epithelial cells induces tenascin-C release mas

Mills J. et al, (2018), INTERNATIONAL JOURNAL OF EXPERIMENTAL PATHOLOGY, 99, A59 - A59

Targeting early changes in the synovial microenvironment: a new class of immunomodulatory therapy?

MIDWOOD K., (2018), Annals of the Rheumatic Diseases

Time to include fine specificity ACPAs in the routine diagnosis and management of rheumatoid

MIDWOOD K., (2018), Arthritis and Rheumatology

How to detect and purify tenascin-C.

Giblin SP. et al, (2018), Methods in cell biology, 143, 371 - 400

Inhibition of endothelial NOTCH1 signaling attenuates inflammation by reducing cytokine-mediated histone acetylation at inflammatory enhancers

Midwood K., (2018), Arteriosclerosis, Thrombosis, and Vascular Biology

Internal affairs: tenascin-C as a clinically relevant, endogenous driver of innate

Midwood K. and Marzeda AM., (2018), Journal of Histochemistry and Cytochemistry

Investigating cell-type specific functions of tenascin-C.

Murdamoothoo D. et al, (2018), 143, 401 - 428

Association of Distinct Fine Specificities of Anti-Citrullinated Peptide Antibodies With Elevated Immune Responses to Prevotella intermedia in a Subgroup of Patients With Rheumatoid Arthritis and Periodontitis.

Schwenzer A. et al, (2017), Arthritis & rheumatology (Hoboken, N.J.), 69, 2303 - 2313

Alternative splicing of tenascin-C modulates cell-matrix interactions during inflammation and disease

Giblin SP. and Midwood KS., (2017), INTERNATIONAL JOURNAL OF EXPERIMENTAL PATHOLOGY, 98, A5 - A5

Mapping tenascin-C interaction with toll-like receptor 4 reveals a new subset of endogenous inflammatory triggers

Midwood K., (2017), Nature Communications

Toll-Like Receptor-4 Signaling Drives Persistent Fibroblast Activation and Prevents Fibrosis Resolution in Scleroderma.

Bhattacharyya S. et al, (2017), Advances in wound care, 6, 356 - 369

Screening for Novel Endogenous Inflammatory Stimuli Using the Secreted Embryonic Alkaline Phosphatase NF-κB Reporter Assay

Midwood K. et al, (2017), Bioprotocols

A unique mode of extracellular matrix mediated immunity: how tenascin-C activates toll-like receptor 4

Zuliani-Alvarez L. et al, (2016), INTERNATIONAL JOURNAL OF EXPERIMENTAL PATHOLOGY, 97, A6 - A6

Tenascin-C at a glance.

Midwood KS. et al, (2016), Journal of cell science, 129, 4321 - 4327

Identification of an immunodominant peptide from citrullinated tenascin-C as a major target for autoantibodies in rheumatoid arthritis.

Schwenzer A. et al, (2016), Ann Rheum Dis, 75, 1876 - 1883

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