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Publications

Identification of TNFR2 and IL-33 as therapeutic targets in localized fibrosis

Izadi D. et al, (2019), Science Advances

Alternative splicing of tenascin-C modulates cell-matrix interactions during inflammation and disease

(2019), MOLECULAR BIOLOGY OF THE CELL

Non-Surgical Periodontal Therapy Plus Short-Term Antibiotic Treatment May Improve Clinical Disease Activity: A Pilot Study in Difficult to Treat Rheumatoid Arthritis

(2019), Arthritis & Rheumatology

Tenascin-C increases lung metastasis by impacting blood vessel invasions.

Sun Z. et al, (2019), Matrix Biol

Treatment of severe periodontitis may improve clinical disease activity in otherwise treatment-refractory rheumatoid arthritis

MIDWOOD KIM., (2019), Rheumatology

Citrullination of fibronectin alters integrin clustering and focal adhesion stability promoting stromal cell invasion.

Stefanelli VL. et al, (2019), Matrix Biol

Response to: 'Potential roles for tenascin in (very) early diagnosis and treatment of rheumatoid arthritis' by Cutolo et al.

Aungier S. et al, (2019), Ann Rheum Dis

Airway Epithelial Cells Generate Pro-inflammatory Tenascin-C and Small Extracellular Vesicles in Response to TLR3 Stimuli and Rhinovirus Infection.

Mills JT. et al, (2019), Front Immunol, 10

Human rhinovirus infection of airway epithelial cells induces tenascin-C release mas

Mills J. et al, (2018), INTERNATIONAL JOURNAL OF EXPERIMENTAL PATHOLOGY, 99, A59 - A59

Targeting early changes in the synovial microenvironment: a new class of immunomodulatory therapy?

MIDWOOD K., (2018), Annals of the Rheumatic Diseases

Time to include fine specificity ACPAs in the routine diagnosis and management of rheumatoid

MIDWOOD K., (2018), Arthritis and Rheumatology

How to detect and purify tenascin-C.

Giblin SP. et al, (2018), Methods in cell biology, 143, 371 - 400

Inhibition of endothelial NOTCH1 signaling attenuates inflammation by reducing cytokine-mediated histone acetylation at inflammatory enhancers

Midwood K., (2018), Arteriosclerosis, Thrombosis, and Vascular Biology

Internal affairs: tenascin-C as a clinically relevant, endogenous driver of innate

Midwood K. and Marzeda AM., (2018), Journal of Histochemistry and Cytochemistry

Investigating cell-type specific functions of tenascin-C.

Murdamoothoo D. et al, (2018), 143, 401 - 428

Alternative splicing of tenascin-C modulates cell-matrix interactions during inflammation and disease

Giblin SP. and Midwood KS., (2017), INTERNATIONAL JOURNAL OF EXPERIMENTAL PATHOLOGY, 98, A5 - A5

Association of Distinct Fine Specificities of Anti-Citrullinated Peptide Antibodies With Elevated Immune Responses to Prevotella intermedia in a Subgroup of Patients With Rheumatoid Arthritis and Periodontitis.

Schwenzer A. et al, (2017), Arthritis & rheumatology (Hoboken, N.J.), 69, 2303 - 2313

Mapping tenascin-C interaction with toll-like receptor 4 reveals a new subset of endogenous inflammatory triggers

Midwood K., (2017), Nature Communications

Toll-Like Receptor-4 Signaling Drives Persistent Fibroblast Activation and Prevents Fibrosis Resolution in Scleroderma.

Bhattacharyya S. et al, (2017), Advances in wound care, 6, 356 - 369

Screening for Novel Endogenous Inflammatory Stimuli Using the Secreted Embryonic Alkaline Phosphatase NF-κB Reporter Assay

Midwood K. et al, (2017), Bioprotocols

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