Cookies on this website

We use cookies to ensure that we give you the best experience on our website. If you click 'Accept all cookies' we'll assume that you are happy to receive all cookies and you won't see this message again. If you click 'Reject all non-essential cookies' only necessary cookies providing core functionality such as security, network management, and accessibility will be enabled. Click 'Find out more' for information on how to change your cookie settings.

Empirical studies attempting to explain tolerance to transplanted tissues have demonstrated a regulatory role for CD4+ T-cells. We here propose that regulatory T-cells mediating transplantion tolerance comprise two sets which can functionally complement each other. The CD4+CD25+ "natural regulator" arises in the thymus, and is preoccupied with self-antigens expressed at sites of inflammation. The second, comprising both CD4+CD25+ (FoxP3+) and CD4+CD25- Tr1-like cells are induced by persistent danger-free antigen in the periphery. The role of these cells is to moderate immune responses to prevent tissue destruction while allowing microbial elimination.

Original publication

DOI

10.1016/j.smim.2003.12.007

Type

Journal article

Journal

Semin Immunol

Publication Date

04/2004

Volume

16

Pages

119 - 126

Keywords

Animals, Antigens, CD, CD4-Positive T-Lymphocytes, DNA-Binding Proteins, Forkhead Transcription Factors, Humans, Immune Tolerance, Models, Immunological, Organ Transplantation, Receptors, Interleukin-2, Self Tolerance, T-Cell Antigen Receptor Specificity, T-Lymphocyte Subsets, T-Lymphocytes, Thymus Gland, Transplantation Immunology